The solar plexus center - The pancreas. EH 625.
Agitation in that body, any violent activity under
stress of temper, intense worry or prolonged irritation will pour a stream of astral energy into and through the solar plexus
center, and will galvanize that center into a condition of intense disturbance. This next affects the stomach, the pancreas,
the gall duct and bladder. EH 38.
Astral body EH 46.
Pancreas Solar Plexus region
Most of the information given in connection with the
pancreas is strictly physiological, and, therefore, out of place here. Suffice it to say, however, that it lies in the abdomen
and is close to the solar plexus (which is the brain of the instinctual animal nature) and is closely concerned with the "mobilization
of energy for physical and mental purposes. It has two secretions, both insulin, one concerned with the digestive processes
and the other known to be vital to the metabolism of sugar. Without sufficient sugar for the cells, no muscle work or nerve
work - essentials in the struggle for existence - are possible." - Berman, Louis, M.D., The Glands Regulating Personality,
p. 93. SM 47.
The dense physical externalization of this center is
the pancreas, with a secondary externalization in the stomach. EH 173.
Diabetes is more definitely the result of wrong inner
desires, and is not so definitely the result of wrong outer desires. These may originate in this life, as I have said above,
or be inherited from a previous existence. In this latter case, the incarnating soul chooses a family in which to be born,
which will endow it with a body having a tendency or a natural predisposition to this disease. EH 312.
Diabetes is more definitely in the class of diseases
which are connected with inner emotional desires and the violent suppressed wish-life of many. Ibid. Adapted.
Soda is taken for the abatement of diabetes...
It is possible to cure diabetes in the beginning by suggestion, if the action is strong enough. Indeed, milk with soda is
always good. Coffee and tea, as well as everything which generates internal alcohol, are not useful. This disease is often
hereditary through a generation; therefore it is impossible to foresee the illness. FW3 536.
The role of Insulin:
Since insulin is the principal hormone that regulates
uptake of glucose into cells (primarily muscle and fat cells) from the blood, deficiency of insulin or its action plays a
central role in all forms of diabetes.
Most of the carbohydrates in food are rapidly digested
to glucose, the principal sugar in blood. Insulin is produced by beta cells in the pancreas in response to rising levels of
glucose in the blood, as occurs after a meal. Insulin makes it possible for most body tissues to remove glucose from the blood
for use as fuel, for conversion to other needed molecules, or for storage. Insulin is also the principal control signal for
conversion of glucose (the basic sugar unit) to glycogen for storage in liver and muscle cells. Lowered insulin levels result
in the reverse conversion of glycogen to glucose when glucose levels fall -- though only in the liver not muscle tissue. Higher
insulin level increase many anabolic ("building up") processes such as cell growth, cellular protein synthesis, and fat storage.
Insulin is the principal signal in converting many of the bidirectional processes of metabolism from a catabolic to an anabolic
If the amount of insulin produced is insufficient,
if cells respond poorly to the effects of insulin (insulin insensitivity or resistance), or if the insulin itself is defective,
glucose is not handled properly by body cells (about 2/3 require it) nor stored appropriately in the liver and muscles. The
net effect is persistent high levels of blood glucose, poor protein synthesis, and other metabolic derangements.
Diabetes mellitus is a medical disorder characterized
by varying or persistent hyperglycemia (elevated blood sugar levels), especially after eating. All types of diabetes mellitus
share similar symptoms and complications at advanced stages. Hyperglycemia itself can lead to dehydration and ketoacidosis.
Longer-term complications include cardiovascular disease (doubled risk), chronic renal failure (it is the main cause for dialysis),
retinal damage with eventual blindness, nerve damage and eventual gangrene with risk of amputation of toes, feet, and even
The most important forms of diabetes are due to decreased
production of insulin (diabetes mellitus type 1, the first recognized form), or decreased sensitivity of body tissues to insulin
(diabetes mellitus type 2, the more common form). The former requires insulin injections, while the latter is generally managed
with oral medication and only requires insulin if the tablets are ineffective.
Patient understanding and participation is vital as
blood glucose levels change continuously, while successfully keeping blood sugar within normal limits has been compellingly
shown to reduce or prevent development of the complications of diabetes. Other risk factors that can require addressing to
reduce complications are: cessation of smoking, optimizing cholesterol levels, maintaining a stable body weight, controlling
high blood pressure and having regular exercise.
The symptoms of CF usually develop
during early childhood. Both lungs and pancreas produce abnormally viscous mucus. The mucus in the lungs can become a growth
medium for bacteria, resulting in chronic respiratory infections and eventual permanent damage to the lung tissue. As lung
function deteriorates, CF patients develop pulmonary hypertension and eventually cor pulmonale. Death usually occurs from
severe infection or heart failure. These thick secretions also obstruct the pancreas, preventing digestive enzymes from reaching
the intestines to help break down and absorb food.
Acute pancreatitis is a rapidly-onset inflammation of the pancreas. Depending on its severity, it
can have severe complications and high mortality despite treatment. While mild cases settle with conservative measures or
endoscopy, severe cases require surgery (often more than one intervention) to contain the disease process. wikipedia.org.